In the Fmr1 knockout mouse model of FXS, expression of Kv1.1 in the brain and neuronal Kv1.1‐mediated currents are significantly reduced in neonates and adults, suggesting FXS‐related Kv1.1 dysregulation that could contribute to ASD phenotypes (Zhu et al., 2018). The gene discussed is KCNA1; the disease is fragile X syndrome.