The results indicated that circ-NOLC1 overexpression in CAOV3 cells significantly induced cell proliferation (Fig. 3A, p < 0.05) and clonal formation (Fig. 3B, p < 0.05); reduced cell apoptosis (Fig. 3C, p < 0.05); and induced cell migration (Fig. 3D, p < 0.05) and invasion (Fig. 3E, p < 0.05), which means that circ-NOLC1 functions as an “oncogene” in ovarian cancer. The gene discussed is NOLC1; the disease is ovarian cancer.