Melanoma, fibrosarcoma, colon cancer, glioblastoma, prostate cancer, and pancreatic ductal carcinoma each grow slower in syngeneic B6 p50−/− than in wild‐type (WT) mice, with M2‐to‐M1 TAM reprogramming and increased tumor T‐cell activation evident in p50−/− tumor hosts [14, 15, 16, 17]. Here, NFKB1 is linked to glioblastoma.