MYL3 and cardiomyopathy: By contrast, we found a set of 29 switch genes shared between the two cardiomyopathies that were all downregulated in the disease (Supplementary Data 2) and appeared functionally enriched (adjusted p-value < 0.05) in the cardiac muscle contraction KEGG pathway, including cardiac-type troponin T2 (TNNT2), myosin light chain 3 (MYL3), the subunits 5A and 7B of the cytochrome c oxidase (COX5A and COX7B), the ubiquinol-cytochrome c reductase core protein 1 (UQCRC1), and cytochrome c1 (CYC1).