Previous GWAS studies identified genetic variants in the lncRNA antisense non-coding RNA gene at the INK4 locus (ANRIL) that was associated with a higher risk of having T2D [39] and coronary artery disease (CAD) [40], therefore, suggesting the involvement of lncRNAs in diabetic CVD complication. The gene discussed is CDKN2A; the disease is coronary artery disorder.