Even if the inhibitory effect of β2GPI on the migration of endothelial cells in vitro was not implied with reduction of cell proliferation or survival (23), the nicked forms, the protein dimers, and the aggregates may play an inhibitory effect on other processes, as suggested by the pathophysiology of the thromboembolic diseases in which these protein-protein interaction forms are produced in vivo. This evidence concerns the gene APOH and Thromboembolism.