Based on this finding, we concurrently applied mSjci and DSS to mice and found that mSjci exerted protective effects on mice with DSS-induced colitis by suppressing proinflammatory Th1 cytokine (IFN-γ) production (Figures 3A,C) and promoting regulatory cytokine (IL-10) secretion (Figures 3B,D). This evidence concerns the gene IFNG and colitis.