UCA1 and gastric cancer: In particular, some HRLs are subjected to a series of posttranscriptional modifications to control gene expression, such as lncRNA UCA1, which can act as a competitive endogenous RNA (ceRNAs) and is involved in miRNA-mediated mRNA destabilization as an endogenous miRNA ‘sponge’ or ‘decoy’ in hypoxia-resistant gastric cancer cells [38].