ABCA1 and diabetic kidney disease: Recently, Ducasa et al. have reported that podocyte-specific deletion of ABCA1 rendered mice susceptible to DKD and the accumulation of mitochondrial cardiolipin, and in mice with DN, an increase in cardiolipin oxidation was observed and a cardiolipin peroxidase inhibitor treatment reversed DKD progression, with improvements in podocyte number [82].