NFKB1 and Duchenne muscular dystrophy: The transcription factor NF-κB, which serves as a main mediator of inflammatory responses, is over-expressed in DMD muscles [14] and its inhibition by different pharmacological compounds or procedures has beneficial effects on the functional, biochemical, and morphological aspects of the muscles and hearts of mdx mice, which is the murine model of DMD [15,16,17,18,19,20,21].