ATP5IF1 and neoplasm: IF1 overexpression promotes mitochondrial hyperpolarization and mitochondrial ROS production and this can be inhibited by MitoQ in tumor cells [31], suggesting that a major effect of MitoQ in the context of LPS-induced NO and cytokine production in the BV2 cells could be via a reduction in ROS generated through hyperpolarization of the mitochondrial membrane.