KLF2, which induces apoptosis, was described to be reduced in the cancer tissues, opposite to what happens to TINCR. Thus, interaction of TINCR lncRNA with STAU1 in cancer cells induces the degradation of KLF2, preventing apoptosis and contributing to the oncogenic potential of gastric carcinoma [96]. This evidence concerns the gene KLF2 and gastric carcinoma.