Viral infections, which have been proposed as triggering factors for T1D [105], were found to induce lnc13 translocation from the nucleus to the cytoplasm, enabling the interaction of STAT1 mRNA with PCBP2, which promotes the signaling events that will ultimately lead to pancreatic beta cell destruction and T1D development [103]. This evidence concerns the gene PCBP2 and type 1 diabetes mellitus.