TP53 and rheumatoid arthritis: The results revealed that after Tenovin‐6 treatment, p53 was activated (Figure 8A), and the cell proliferation, viability and resistance to apoptosis and cell cycle arrest of the RA‐FLSs were significantly reduced compared to those of the Evs‐IN‐treated RA‐FLSs (Figure 8B‐H).