As in E. coli endotoxin induced acute lung injury, the anti‐inflammatory effect of human‐MSC EVs was only partially explained by the transfer of keratinocyte growth factor (KGF) and angiopoietin‐1 (Ang‐1) mRNA into injured cells,9, 22 we hypothesized that other transferable genetic materials such as microRNAs23, 24 could be involved in the therapeutic benefit of MSC EVs in MDR‐PA inoculation pneumonia. Here, FGF7 is linked to susceptibility to pneumonia measurement.