DAAM1 and breast cancer: Cell motility depends on the dynamics of actin filaments and pseudopodia, which are involved in adhesion to the extracellular matrix (ECM), guidance towards chemoattractants, degradation of ECM, transduction of extracellular signal, output of forces, etc.2, 3We have demonstrated that dishevelled‐associated activator of morphogenesis 1 (Daam1), a member of formin family proteins, mediates ECM‐induced invadopodia extension and cell migration in breast cancer (BrCa).4