KLRG1 and infection: TEM cell subsets, however, were mainly responsible for the difference between LCMV Armstrong and MCMV-GP33 infections, with Ly6C+KLRG1−CD127+ cells being more abundant in LCMV Armstrong and KLRG1+CX3CR1+NKG2A+ GP33-specific CD8+ TEM cells defining the phenotype after MCMV-GP33 infection.