TLR7 and systemic lupus erythematosus: STAT4 is also largely dispensable for the regulation of autoimmune AFC, GC, and Th cell responses, autoantibody production and SLE manifestations in Spt autoimmune-prone Sle1b and SLE-prone FcγRIIB−/− mice, and TLR7-induced, SLE-prone Sle1b mice.