Another work identified the transcriptome profile of left and right atrial appendages of patients with AF versus controls and identified NPPA and its antisense as potential regulators of muscle contraction in AF and moreover RP11‐99E15.2 and RP3‐523K23.2 which could modulate extracellular matrix binding and transcription of HSF2 targets, respectively.136. The gene discussed is HSF2; the disease is atrial fibrillation.