It can induce apoptosis in human SGC-7901 gastric cancer cells by acting on the stress response, p53-dependent mitochondrial-mediated, caspase-8/-3-dependent, and PI3k/Akt pathways [42]; apoptosis, cell cycle arrest at S phase, and the modulation of the expression of 21 proteins were observed and, in particular, the upregulation of 10 proteins, including RBBP4, associated with cell cycle arrest and the downregulation of 11 proteins, including RUVBL1, NPM, HSP90AB1, and GRP78, involved in apoptosis and stress response. The gene discussed is AKT1; the disease is gastric cancer.