In addition, the knockdown of XIAP attenuates anchorage-independent cell growth [149], suggesting that the anchorage-independent growth of bladder cancer can be modulated by the XIAP/miR-200/EGFR/EMT axis (see Section 3.3.2 for EGFR/EMT relationship) (Figure 2 and Table 2). This evidence concerns the gene EGFR and urinary bladder carcinoma.