AGTR1 and glioblastoma: A further study on the miRNA-AGTR1 relationship and the molecular mechanism of AGTR1-mediated signaling showed that miR-155-5p depresses EMT process and anchorage-independent growth by attenuating the activity of NF-κB via targeting AGTR1 in glioblastoma [144] (Figure 2 and Table 2).