Further, it was observed that overexpression of miR-424-5p causes a reversal of EMT status, induces anoikis, and suppresses the tumorigenicity of hepatocellular carcinoma by targeting beta-catenin-interacting protein 1 (CTNNBIP1, also known as ICAT), an endogenous β-catenin repressor [155] (Figure 2 and Table 2). The gene discussed is CTNNBIP1; the disease is hepatocellular carcinoma.