FH is an autosomal-dominant pathology, identified in all races and ethnic groups and determined by mutations of the low density lipoprotein receptor (LDLR), apolipoprotein B (APOB), proprotein convertase subtilisin/kexin type 9 (PCSK9) genes, and it is considered a cause of premature coronary atherosclerotic disease [4, 5]. Here, APOB is linked to familial hyperaldosteronism.