KCNA1 and atrial fibrillation: Following the discovery of Kv1.1 expression in mouse cardiomyocytes, electrophysiology studies in mice demonstrated that the lack of Kv1.1 channel subunits impairs repolarization leading to prolonged atrial action potentials and significantly increased risk of inducible atrial fibrillation (Glasscock et al., 2015; Si et al., 2019).