HOXA9 and acute myeloid leukemia: Overexpression of HOXA9 in murine marrow cells leads to expansion of HSCs and committed progenitors that likely undergo transformation to acute myeloid leukemia (AML) with co-expression of Meis1 and Pbx3, emphasizing the gene’s key role in blood physiology and pathology (Dassé et al. 2012)