LDLR and familial hyperaldosteronism: In total, 110/201 or 55% of these patients were confirmed to have FH by genetic testing (57 males and 53 females); 108 were identified with heterozygous FH and two as homozygous FH, with the majority of pathogenic variants identified in the LDL receptor gene.5 None of the patients within this paediatric cohort had cardiovascular events either at baseline or during the follow-up period.5