In models of infection with the above fungal pathogens, some studies have shown that Th17/IL-17 axis mediate resistance, while others have shown that they promote disease pathology (Deepe and Gibbons, 2009; Loures et al., 2009; Loures et al., 2009; Wüthrich et al., 2011; Nanjappa et al., 2012; Wu et al., 2013; Wang et al., 2014; Pino-Tamayo et al., 2016; Puerta-Arias et al., 2016; Ketelut-Carneiro et al., 2019). This evidence concerns the gene IL17A and infection.