Wang et al. reported that interferon-γ released from CD8+ T cells downregulates the expression of SLC3A2 and SLC7A11 and, as a consequence, promotes tumor cell lipid peroxidation and ferroptosis [35], suggesting that the selective killing ability of CD8+ T cells for tumor cells is partially due to ferroptosis induced by interferon-γ. The gene discussed is SLC7A11; the disease is neoplasm.