To further investigate the functions of ERLIN2 in LUAD, we conducted GSEA using TCGA data, which showed that GPI anchor biosynthesis, hematopoietic cell lineage, intestinal immune network for IgA production, allograft rejection, graft versus host disease, asthma, cytokine receptor interaction, type I diabetes mellitus, ribosome, and NKC-mediated cytotoxicity in KEGG are differentially enriched in the ERLIN2 high-expression phenotype. The gene discussed is CD79A; the disease is glycogen storage disease VI.