To ensure correspondence of behaviors seen in CD103KO mice to human ASD and/or ADHD, as well as to identify possible mechanistic targets to modulate such behaviors, we therefore quantified NeuN, Drebrin, Synaptophysin, GAPDH, and eIF4E in separate female and male wt and CD103 cohorts. This evidence concerns the gene GAPDH and attention deficit-hyperactivity disorder.