AXL and viral infectious disease: Viral infection was significantly enhanced in HEK293T cells overexpressing human ACE2 or human AXL, but not in HEK293T cells expressing the empty vector (Fig. 6a–c) or murine or rhesus macaque AXL (Supplementary information, Fig. S3h), reproducing the results observed using the SARS-CoV-2 virus pseudotype and confirming that both ACE2 and AXL are SARS-CoV-2 receptors.