This assertion is based on experiments showing that post-recall infusion of AP5 and TCN, but not of RO, impaired extinction memory retention lastingly and induced the recovery of avoidance in a time-dependent manner whereas pre-recall administration of AP5 and RO, but not of TCN, rendered the reactivated extinction memory trace resistant to the amnesia caused by mTOR inhibition. This evidence concerns the gene MTOR and amnesia.