IL18 and colitis: In particular, we assessed the expression of TLR2, 3, 4, 5 and 7 and antimicrobial peptides (Defα24, RELMβ and RegIIIγ), according to their high expression within the gastrointestinal tract [40] and their implication in states of dysbiosis [41,42,43] as well as pro- (IL-6, INFɣ, IL-18, IL-12p40) and anti-inflammatory cytokines (IL-10), implicated in the development of colitis [44].