DNM1 and Alzheimer disease: This is in line with our observation of higher Aβ42 content in Dynamin enriched compartments, and is also consistent with the previous reports of increased Aβ content in both synaptic and endosomal compartments during the molecular progression of AD (Abramov et al., 2009; Gouras et al., 2010; Pickett et al., 2016; Sannerud et al., 2011, 2016).