It is the case of the Wnt pathway, that has been shown to contribute to renal fibrosis, in in vivo and in vitro models (Satoh et al., 2012; Kawakami et al., 2013; LeBleu et al., 2013); of Nuclear Factor kappa-light-chain-enhancer of activated B cells (NF-κB) and Mitogen Activated Protein(MAP) kinases that further enhance the inflammatory state (Park et al., 2017); of mTOR, that has been associated with enhanced TGF-β expression (Cheng and Hao, 2017). Here, TGFB1 is linked to renal fibrosis.