The results showed that bladder cancer, cell cycle, DNA replication, glycosaminoglycan biosynthesis chondroitin sulfate, homologous recombination, glycan biosynthesis, nucleotide excision repair, p53 signaling pathway, pyrimidine metabolism, and spliceosome were enriched in the high FOXD1 expression group (Figure 6). This evidence concerns the gene TP53 and urinary bladder cancer.