Since the histological features and cognitive impairments seen in αSyn fibril-injected mice were suppressed by Fabp3 deletion, it is likely that FABP3 participates in αSyn pathology in not only nigral dopaminergic neurons but also other regions whose cell subpopulations highly express FABP3, such as septal GABAergic neurons. The gene discussed is FABP3; the disease is Cognitive impairment.