In experimental hamsters, an infusion of GLP1 and GLP2 causes an initial increase in lipid absorption and increased plasma concentrations of TRL-apo B48 in plasma and this response in terms of the increased postprandial lipid response has been reported to be enhanced under the condition of insulin resistance, such as in type 2 diabetes [40]. The gene discussed is INS; the disease is type 2 diabetes mellitus.