Conversely, AAC2 is significantly upregulated in BRCA, kidney chromophobe cancer (KICH), LUSC, and uterine corpus endometrial carcinoma (UCEC), at variance with AAC3 significantly upregulated in CHOL, KICH, and PRAD (Figure 1). This evidence concerns the gene SLC25A6 and uterine corpus endometrial carcinoma.