IFNG and myocarditis: Notably, T. cruzi infection in the heart enhances the production of IFN-γ, which increases T cell migration and the establishment of myocarditis by promoting the expression of chemokines (CCL5, CCl2, CXCL10 and CXCL9) and chemokine receptors, intercellular adhesion molecules and the vascular cell adhesion molecule [96,97].