Or, it could be possible that corticosterone‐driven insulin resistance (Dallman et al., 1995; Jimeno et al., 2018; Morakinyo et al., 2019; Strack et al., 1995; Tadaishi et al., 2018) could increase plasma glucose which would then stimulate the release in insulin, and that this could be modulated by AR blockade. The gene discussed is AR; the disease is Insulin resistance.