Or, it could be possible that corticosterone‐driven insulin resistance (Dallman et al., 1995; Jimeno et al., 2018; Morakinyo et al., 2019; Strack et al., 1995; Tadaishi et al., 2018) could increase plasma glucose which would then stimulate the release in insulin, and that this could be modulated by AR blockade. This evidence concerns the gene INS and Insulin resistance.