TMPRSS2 and viral infectious disease: When re‐plotting the results obtained in Figure 2d as a function of the absolute amount of ACE2 measured for these same samples by ELISA (Figure 2f), we observed that EVs from cells overexpressing the full length TMPRSS2 together with ACE2 were more efficient at inhibiting SARS‐CoV‐2‐S‐pseudotyped viral infection than those from cells overexpressing ACE2 alone (Figure 2h).