Expression of a chimera consisting of the TGF-β receptor type II (TGFBR2) extracellular and transmembrane domain fused to the NKG2D intracellular domain on NK-92 cells caused tolerance to the TGF-β signaling and improved chemoattraction to TGF-β-secreting tumor cell lines (102). Here, TGFB1 is linked to neoplasm.