UCP2 and Parkinson disease: In the 1-methyl-4-phenyl-1,2,5,6 tetrahydropyridine (MPTP) mouse model of PD, AG protected from neuronal death in the SNpc, as displayed by the normalized B-cell lymphoma 2 (Bcl-2)/Bax ratio and lowered caspase-3 activity, stimulated the neuronal activity, elevated the production of multiple LCFAs, for instance palmitic acyl CoA (C16:0), and improved the mitochondrial respiration by activating the ROS-buffering, mitochondrial uncoupling protein 2 (UCP2).