Previously, we used the transgenic zebrafish model to demonstrate that liver‐specific overexpression of HBx and src oncogene promotes hepatocarcinogenesis, including steatosis and fibrosis in the earlier stages and hyperplasia, dysplasia, and HCC in the later stages,13 diet‐induced obesity can accelerate hepatocarcinogenesis.14 Here, SRC is linked to hepatocellular carcinoma.