LPS‐induced ALI demonstrated pathological features similar to human ALI /ARDS,22 including lung inflammation reflected by leukocyte and neutrophil recruitment (Figure 3B,C), capillary barrier dysfunction by alveolar protein influx (Figure 3D), and plasma leakage (Figure 3E), and over‐production of inflammatory mediators, for example, IL‐1β (Figure 3F) or IL‐6 (Figure 3G), and tissue injury by pathological scores (Figure 4). This evidence concerns the gene IL1B and inflammation.