Gi-like defects were diagnosed in 38 patients (32.2%), complex disorders in 33 cases (28.0%), TxA2 pathway defects in 14 (11.9%), diminished procoagulant COAT platelets in 10 (8.5%), collagen receptor defects in eigth (6.8%), α-granule disorders in six (5.1%), dense granule disorders in five (4.2%), and Glanzmann thrombasthenia in four patients (3.4%). Here, ITGA2 is linked to Glanzmann thrombasthenia.