Total granulocytes and their activated CD11b+ subset were also increased in COVID-19 patients (69.0% vs. 56.84%, p < 0.001 and 72.33% vs. 46.12% of controls, respectively) at the expense of a statistically significant downregulation of the CD11b− granulocyte counterpart (15.74% vs. 47.29% of controls; p < 0.05). This evidence concerns the gene ITGAM and COVID-19.