In parallel, we checked the expression of solute carrier transporters present in the agilent platform (Figure 6B), and, in all prostate cancer cell lines analyzed upon MALAT1 depletion, we found a statistically significant downmodulation exclusively of SLC25A1, a mitochondrial citrate transporter mediating the exchange of mitochondrial citrate with cytosolic malate [32,33,34]. This evidence concerns the gene MALAT1 and prostate carcinoma.