Although the pathogenesis of MGN is not fully understood, recent studies revealed the autoantibodies to the M-type receptor for phospholipase A2 receptor 1 (PLA2R1) [28], thrombospondin type-1 domain-containing 7A (THSD7A) and neural epidermal growth factor-like 1 protein (NELL-1) as the pathogenesis of MGN [28,29,30]. Here, THSD7A is linked to idiopathic membranous glomerulonephritis.