TNFSF11 and idiopathic membranous glomerulonephritis: Based on the report, Li et al. analyzed the expression of plasma and kidney miR-217 in MGN patients and reported the decreased miR-217 expressions in plasma and kidney tissues in MGN patients compared to control patients [33], and also demonstrated that the downregulation of miR-217 may cause podocyte apoptosis via targeting TNF superfamily member 11 (TNFSF11).