However, other pancreatic adenocarcinoma studies have implicated neutrophils in shorter survival: high levels of CD66b positive tumor infiltrating cells were associated with shorter overall survival,18 poor prognosis “squamous” subtype tumors were shown to recruit neutrophils via a p63‐dependent mechanism,27 and depletion of CXCR2 or Ly6G positive cells (which includes neutrophils and myeloid derived suppressor cells) suppressed metastasis.29 This evidence concerns the gene TP63 and neoplasm.