TGFBR1 and endometriosis: For example, our preliminary investigations demonstrated that miR‐96‐5p was able to interact with the TGF‐β/SMAD signalling pathway in endometrial cells via direct targeting of TGFBR1, whereas miR‐34c‐5p could suppress the progression of EMT through targeting the Notch signalling pathway in endometriosis.44, 45