For example, our preliminary investigations demonstrated that miR‐96‐5p was able to interact with the TGF‐β/SMAD signalling pathway in endometrial cells via direct targeting of TGFBR1, whereas miR‐34c‐5p could suppress the progression of EMT through targeting the Notch signalling pathway in endometriosis.44, 45. This evidence concerns the gene TGFB1 and endometriosis.